By Susan B. Waltzman, J. Thomas Roland
The moment variation of Cochlear Implants presents a accomplished evaluate of the state of the art strategies for comparing and choosing the cochlear implant candidate. transparent descriptions of surgical strategies consultant the reader via implantation tactics, and chapters tackle very important matters reminiscent of speech creation, language improvement, and schooling in implant recipients.
This moment variation features:
- New chapters at the genetics of listening to loss, sound processing, binaural listening to, and electroacoustic stimulation
- Complete dialogue of the newest advances in evaluate tactics, surgical procedure, programming equipment, speech processing innovations, and extra
- Precise, easy-to-follow tables and figures increase comprehension of the fundamental technological know-how, study and scientific ideas lined in the textual content
- Coverage of the scientific and surgical issues of cochlear implantation
- Insights from an interdisciplinary group of specialists in otolaryngology, audiology, the elemental sciences, speech pathology, and education
Ideal for studying and reference, Cochlear Implants synthesizes the foremost details wanted by means of practitioners, researchers, and scholars in a variety of disciplines. Readers will make the most of either the scope and thoroughness of this authoritative reference.
Dr. Roland venerated in most sensible medical professionals 2012 factor of big apple Magazine
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Extra info for Cochlear Implants
98,99 Although deafferentation during cochlear development results in widespread neural loss in the CN, similar cochlear damage after the onset of hearing results in atrophic changes but not neural loss. Given that most forms of SNHL occur following the onset of hearing,100 – 102 these results imply that the major transneuronal effects of deafness are atrophic, with relatively small amounts of neural loss. An alternative form of deafening is provided by the administration of ototoxic drugs. , amikacin, neomycin, kanamycin, ethacrynatic acid) are known to cause a loss of hair cell receptors and deafness7,103 – 105 as described above.
Jervell and Lange-Nielsen (1957) originally reported four siblings with profound congenital SNHL, syncopal attacks, sudden death, and a prolonged corrected QT interval (QTc) shown on electrocardiography. The deafness is usually profound; vestibular dysfunction has not been documented (Cusimano et al, 1991). Cardiogenic syncope is common and sudden death occurs in 70% of untreated cases due to ventricular arrhythmias (torsades de pointes), which are usually precipitated by a sudden increase in autonomic activity (Cusimano et al, 1991).
Mitochondrial mutations and hearing loss: paradigm for mitochondrial genetics. Am J Hum Genet 62:15 – 19 Fischel-Ghodsian N. (1999). Mitochondrial deafness mutations reviewed. Hum Mutat 13:261 – 270 Fischel-Ghodsian N. (2003). Mitochondrial deafness. Ear Hear 24:303– 313 Fukushima K, Sugata K, Kasai N, et al. (2002). Better speech performance in cochlear implant patients with GJB2-related deafness. Int J Pediatr Otorhinolaryngol 62:151 – 157 Gordon N. (1994). The long Q-T syndromes. Brain Dev 16:153 – 155 Gorlin RJ, Toriello HV, Cohen MM, eds.