By David M. Cline, John Ma, O. John Ma, Gabor Kelen, Steven Stapczynski
This article makes use of tables, charts and an overview layout to summarize the most important issues of pathophysiology, analysis, and administration stipulations encountered within the Emergency division. It gains insurance of medical protocols, fluid resuscitation and pelvic discomfort.
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Additional resources for Emergency Medicine: A Comprehensive Study Guide, Companion Handbook
The calculation is 6 mg times weight in kilograms; fill to 100 mL with D5W. , an infusion running at 1 mL/h = 1 µg/kg/min, or 5 mL/h = 5 µg/kg/min). Epinephrine is the only drug proven effective in cardiac arrest. It is indicated in pulseless arrest and in slow rates that are hypoxia induced and unresponsive to oxygenation and ventilation. 2 mg/kg of a 1:1000 concentration) subsequently. 1 mg) after adequate oxygenation and ventilation are ensured. Sodium bicarbonate is no longer recommended as a first line resuscitation drug.
They include CNS delete (weakness, cramps, and hyporeflexia), gastrointestinal (GI, ileus), cardiovascular (dysrhythmias, worsening of digoxin toxicity, and hypotension or hypertension, U waves and ST depression, and prolonged QT), and renal disorders (metabolic alkalosis and worsening hepatic encephalopathy), and, last, glucose intolerance can also develop. Diagnosis and Differential The most common cause is the use of loop diuretics. Table 4-3 lists the causes. 25 meq/L. Patients should be monitored continuously for dysrhythmias.
Hyperphosphatemia occurs mainly with renal failure and can be treated with hydration, acetazolamide, or dialysis. Chloride abnormalities usually occur in association with other metabolic disorders, such as metabolic alkalosis (hypochloremia) or dehydration (hyperchloremia). Treatment for chloride disorders is the correction of the volume deficit with NS, the acid-base abnormality, or the potassium deficit. ACID-BASE PROBLEMS The lungs and kidneys primarily maintain the acid-base regulation. Acidosis is due to gain of acid or loss of alkali; causes may be metabolic (fall in serum HCO3–) or respiratory (rise in PCO2).