By Mai P. Hoang, Martin C. Mihm Jr.
Melanocytic Lesions: A Case established method offers a concise but finished instruction on the right way to diagnose universal in addition to difficult and hard melanocytic lesions. within the first eleven chapters, each one entity is illustrated through an exact case; by way of dialogue of the way the prognosis is reached, of the histologic differential diagnoses and of diagnostic pitfalls and ends with bulleted instructing issues. Pertinent and recent references are integrated on the finish of every bankruptcy. The latter chapters conceal present microstaging and category of cancer, ancillary concepts together with immunohistochemistry in addition to on hand molecular assays and molecular specified treatment. All figures and glass slides of the mentioned circumstances are hosted on-line for simple viewing and entry. Melanocytic Lesions: A Case dependent technique will serves as an invaluable source for pathologists, dermatologists and researchers facing melanocytic lesions.
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Extra resources for Melanocytic Lesions: A Case Based Approach
The type-C cells stain only with S100 protein. Atypical architectural features such as poor circumscription, single cell proliferation, asymmetry, and irregularly confluent nests and cytologic atypia can be seen as a small subset of compound nevi (Urso et al. 2005). Mitoses have also been reported in benign nevi (Jensen et al. 2007). Extension of nevus cells around adnexal structures is commonly seen in the head and neck region; thus, this should not be interpreted as features of congenital onset.
Mihm Jr. 2 Variations on the Acquired Nevi 51 Case 2I Clinical History A 15-year-old female with a changing nevus on her forearm Microscopic Diagnosis Beneath a scale crust there is psoriasiform hyperplasia and intraepidermal edema (spongiosis). The scale crust contains remnant of neutrophils admixed with parakeratosis and protein-rich fluid. The superficial dermal inflammatory infiltrate is associated with marked edema of the papillary dermis and multifocal parakeratosis (Fig. 27). The inflammatory infiltrate essentially spares the nevus component (Fig.
Int J Dermatol. 1996;35(4):229–39. Betti R, Inselvini E, Palvarini M, Crosti C. Agminated intradermal Spitz nevi arising on an unusual speckled lentiginous nevus with localized lentiginosis: a continuum? Am J Dermatopathol. 1997;19(5):524–7. Bhawan J, Purtilo DT, Riordan JA, Saxena VK, Edelstein L. Giant and ‘granular melanosomes’ in leopard syndrome: an ultrastructural study. J Cutan Pathol. 1976;3(5):207–16. Bolognia JL. Reticulated black solar lentigo (‘ink spot’ lentigo). Arch Dermatol. 1992;128(7):934–40.