Download Respiratory Disease: A Case Study Approach to Patient Care by James R. Dexter MD FACP FCCP, Robert L. Wilkins PhD RRT PDF

By James R. Dexter MD FACP FCCP, Robert L. Wilkins PhD RRT FAARC, Philip M. Gold MD FACP FCCP

Now edited through a pulmonologist, the third version remains to be some of the most well-written texts for college kids studying to appreciate the review and remedy of sufferers with respiration ailment. every one bankruptcy starts with a historical past of chosen problems, through a case learn with questions and solutions designed to stimulate serious considering abilities.

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Extra info for Respiratory Disease: A Case Study Approach to Patient Care

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What probably explains Mr. B’s cool and cyanotic extremities? Mr. B’s cool and cyanotic extremities are probably related to poor cardiac output. Poor perfusion of the extremities causes them to become cool to the touch, and the cyanosis is related to the presence of deoxygenated hemoglobin. INTERPRETATION OF CLINICAL LAB DATA Laboratory data are used for general evaluation of the patient, to diagnose specific problems, and to determine the effectiveness of therapy. This section focuses on those tests that are most frequently ordered for patients with cardiopulmonary disease.

Acute injury to the myocardium causes the heart muscle cells to release excessive amounts of enzymes into the circulating blood. For example, creatine phosphokinase (CPK) and troponin enzymes are usually elevated within hours of a heart attack. If the chest pain is not due to ischemia of the heart muscle, the enzymes will not be released into the blood. 3. S Arterial Blood Gases Another common test done to evaluate patients with cardiopulmonary disease is the arterial blood gas (ABG). This test measures the ability of the lungs to oxygenate the blood and remove carbon dioxide from it.

In about 48 to 72 hours, the sample will reveal the identity of the organisms present in the specimen and the antibiotics to which they are most sensitive. Cardiac enzymes are measured in patients suspected of having a myocardial infarction. Acute injury to the myocardium causes the heart muscle cells to release excessive amounts of enzymes into the circulating blood. For example, creatine phosphokinase (CPK) and troponin enzymes are usually elevated within hours of a heart attack. If the chest pain is not due to ischemia of the heart muscle, the enzymes will not be released into the blood.

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