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By C. Srikant

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Acta Endocrinol. 80 (suppl. 200): 1-41. 25. ‚ Yamamoto‚ M. and Fujii-Kuriyama‚ Y. (2001) Defective development of secretory neurones in the hypothalamus of Arnt2-knockout mice. Genes Cells 6: 361-374. 26. ‚ Edlund‚ T. and Edlund‚ H. (1994) Insulin-promoter-factor 1 is required for pancreas development in mice. Nature 371: 606-609. 27. ‚ Bolivar‚ F. F. (1980) Identification of a putative hypothalamic mRNA coding for somatostatin and of its product in cell-free translation. Biochem. Biophys. Res.

Endocrinology 102: 523-530. 52. H. M. (1991) Regulation of islet somatostatin secretion and gene expression: selective effects of adenosine 3'‚5'monophosphate and phorbol esters in normal islets of Langerhans and in a somatostatinproducing rat islet clonal cell line 1027-B2. Endocrinology 128: 1754-1762. 53. J. F. (1980) Identification of prosomatostatin in pancreatic islets. Proc. Natl. Acad. Sci. USA 77: 2410-2414. 54. ‚ Kamps‚ M. and Montminy‚ M. (1995) The pancreatic islet factor STF-1 binds cooperatively with Pbx to a regulatory element in the somatostatin 15 promoter: Importance of the FPWMK motif and the homeodomain.

PreproCST mRNA appears at day post-natal 15 34 and reaches a maximum level during the second post-natal week (between P15 and P20). This pattern of expression correlates with the maturation of cortical interneurons. The regulation of preprocortistatin mRNA accumulation has been investigated in various paradigms underlying again the differences between the two peptides. PreproSST mRNA increases its steady-state concentration four fold upon kainate injection in the dentate gyrus and CA1, whereas preprocortistatin mRNA does not respond to kainate, indicating that CST and SST respond to different signals [16].

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